Objective To explore the specific pathogenic role of glycation products in normal kidney.

目的为探讨糖基化产物对正常肾脏的特异性损害作用。

Conclusion: Puerariae can significantly inhibit glycation reaction in rats induced by D-galactose.

结论：葛根素对d -半乳糖诱导的大鼠糖基化反应具有明显的抑制作用。

The glycation effect caused by glyoxal, methylglyoxal and other saccharide derivatives is toxic to cells.

糖代谢的衍生物甲基乙二醛等可通过糖化作用产生细胞毒性。

RESULTS Inactivation of esterase was induced by glycation, carbamylation and steroid in a concentration .

结果糖化、氨甲酰化和类固醇诱导酯酶失活的作用呈浓度依赖性，类固醇失活效应最快。

AIM To establish a rapid method for screening advanced glycation end product (AGE) cross links breaker in vitro.

目的建立一种快速、体外筛选晚期糖基化终产物(age)交联结构裂解剂的方法。